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Chance and the prepared mind in drug discovery
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The death versus alcohol relationship creme became more consistent with a straight line, linear dose response, meaning more alcohol, more death: no protection at low levels of handen consumption. So: no apparent benefit of light to moderate drinking after all, when you use better comparison groups. Although these results are not what the majority of drinking adults may desire to believe, the public deserves to hear and to read in more complete and balanced detail the ever-growing evidence that drinking alcohol is very unlikely to improve their health, please consider volunteering. Image credit: joel Herzog via unsplash. Image has been modified.
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Seattle health, medical, life Sciences & biotech Groups
If you look at studies of smokers, sometimes you see higher mortality rates among those who quit smoking, compared to those that continue smoking. Because the reason they quit smoking is because they got sick. So, of course, sick people die more often than less sick people. Thats why when you classify someone as a face non-smoker in a study, you have to make sure theyre a lifelong nonsmoker and not just a non-smokersince last tuesday. Yet, unbelievably, thats not what they do in most alcohol studies, where instead they misclassify former drinkers as if they were peeling lifelong abstainers. And, look, individuals with poorer health are more likely to cut down or stop drinking completely, thereby making current drinkers look good in comparison to those who drink zero, because some of the abstainers are just abstaining because they got sick and stopped. So, what if you went back to all those studies and corrected the misclassifications, separated out the former drinkers from the lifelong abstainers? We didnt know until now. They, indeed, found drinker misclassification errors all too common, plaguing three quarters of the studies, and when they controlled for that, the j-shaped curve disappeared.
Machine learning applications in cancer prognosis and
Sure, there are statin drugs, but alcoholic beverages dont require a prescription, are far cheaper, and certainly more enjoyable. Is moderate drinking really protective? Or, is there just something about people who abstain completely from alcohol that puts them in a higher risk category? The reason we suspect something fishy is going on is that abstainers seem to be at higher risk of a whole swath of diseases including, ironically, liver cirrhosis. Compared to lifelong abstainers—those who have never touched the stuff—men and women drinking a little appear to have less liver cirrhosis. How could a little drinking be linked to lower rates of liver cirrhosis? Well, lets think about. What makes more sense, that drinking leads to less liver cirrhosis, or liver cirrhosis leads to less drinking? In lenzen other words, reverse causation: the so-called sick quitter effect.
And, if you look at subclinical markers of atherosclerosis, like the waar thickening of the wall of your carotid arteries in your neck, those that abstain from alcohol completely seem to be at the lowest risk. And, the same with coronary calcium scores, where, in general, the lower the alcohol consumption, the lower the risk. And, alcohol bumps our blood pressure up a bit as well, which would be expected to raise, not lower, our cardiac risk. So, where did we get this idea that alcohol was good for us? From the famous J curve. If you follow large populations of people over time, in general, the more people drink, the higher their risk of dying prematurely. But the lowest risk—those who tend to live the longest—are not those who drink zero, the abstainers, but those who drink moderately, like one drink a day. Thats why you get some folks recommending that physicians should counsel lifelong nondrinkers to take up the habit.
Seattle technology, science & Engineering Groups
Below botulinum is an approximation of this videos audio content. To see any graphs, charts, graphics, images, and"s to which. Greger may be referring, watch the above video. Why do we not see the corporate interests of the alcohol industry as clearly as we see those of the tobacco industry? Well, the alcohol industry has waged a sophisticated and successful campaign over the last few decades undermining perceptions of the extent of alcohol-related harms to health by framing the argument as a balance of benefits and harms. Yes, alcohol may be an intoxicating carcinogen, increasing cancer risk, but what about reducing heart disease risk? Policymakers hesitate to introduce effective alcohol policies, or even to support the addition of warning labels, for fear they might undermine or contradict any possible health benefits of alcohol use. After all, alcohol consumption clearly raises hdl, the supposed good cholesterol, but, sadly, as i already explored, hdl is no longer considered protective, based in part on so-called Mendelian randomization studies, where having a high hdl your whole life doesnt appear to help, whereas. So, the boost in hdl from alcohol may not matter.